Search results for "Pancreatic polypeptide"

showing 6 items of 6 documents

Absence of cardiovascular autonomic dysfunction and vagal pancreatic impairment in idiopathic achalasia of the oesophagus

2007

Abstract  Extra-oesophageal autonomic dysfunction in idiopathic achalasia is not well documented, due to contradictory results reported. We aimed to study the cardiovascular and pancreatic autonomic function in patients with idiopathic achalasia. Thirty patients with idiopathic achalasia (16M/14F; 34.5 ± 10.8 years) and 30 healthy volunteers (13M/17F; 34.8 ± 10.7 years) were prospectively studied. Age >60 years and conditions affecting results of autonomic evaluation were excluded. Both groups underwent the sham feeding test and plasmatic levels of pancreatic polypeptide (PP) were determined by radioimmunoassay (basal, at 5, 10, 20 and 30 min). Cardiovascular parasympathetic (deep breathing…

AdultMaleAutonomic functionmedicine.medical_specialtyAdolescentPhysiologyDiaphragmatic breathingAchalasiaAutonomic Nervous SystemPancreatic PolypeptideEatingEsophagusInternal medicinemedicineHumansPancreatic polypeptideEndocrine and Autonomic Systemsbusiness.industryGastroenterologyVagus NerveRadioimmunoassayMiddle Agedmedicine.diseaseSham feedingEsophageal AchalasiaBlood pressureTasteAnesthesiaCardiologyMasticationFemaleIdiopathic achalasiabusinessNeurogastroenterology & Motility
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Autonomic dysfunction in patients with achalasia.

1995

It has been previously shown that patients with achalasia may have motor abnormalities of the stomach, small bowel and biliary system. This study investigates whether a disturbance of extraintestinal autonomic function occurs. Autonomic function studies were performed in 15 patients with achalasia and 15 age- and sex-matched healthy controls. Pupillo-grams were obtained during darkness, light exposure and after pilocarpine administration. Cardiovascular function studies included determinations of heart rate variation during deep breathing and orthostasis. In addition, we determined blood pressure changes in response to sustained handgrip, cold exposure and orthostasis. Neurohormonal functio…

AdultMalemedicine.medical_specialtyPhysiologyAchalasiaDiaphragmatic breathingAutonomic Nervous SystemPancreatic PolypeptideInternal medicineReflexmedicinePancreatic polypeptideHumansEndocrine and Autonomic Systemsbusiness.industryStomachNeuropeptidesGastroenterologyHemodynamicsPupilMiddle Agedmedicine.diseaseSham feedingEsophageal AchalasiaAutonomic nervous systemmedicine.anatomical_structureBlood pressurePilocarpineAnesthesiaCardiologyFemalebusinessmedicine.drugNeurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society
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Pancreatic polypeptide stimulates mouse gastric motor activity through peripheral neural mechanisms

2016

Background Pancreatic polypeptide (PP) is supposed to be one of the major endogenous agonists of the neuropeptide Y4 receptor. Pancreatic polypeptide can influence gastrointestinal motility, acting mainly through vagal mechanisms, but whether PP acts directly on the stomach has not been explored yet. The aims of this study were to investigate the effects of PP on mouse gastric emptying, on spontaneous tone of whole stomach in vitro and to examine the mechanism of action. Methods Gastric emptying was measured by red phenol method after i.p. PP administration (1–3 nmol per mouse). Responses induced by PP (1–300 mmol L−1) on gastric endoluminal pressure were analyzed in vitro in the presence o…

Male0301 basic medicinemedicine.medical_specialtyPhysiologyGastric emptyingMuscarinic AntagonistsBiologyEndocrine and Autonomic SystemMice03 medical and health sciencesOrgan Culture Techniques0302 clinical medicineInternal medicineMuscarinic acetylcholine receptormedicineAnimalsPancreatic polypeptidePeripheral NervesPancreatic polypeptideReceptorAntrumDose-Response Relationship DrugGastric emptyingEndocrine and Autonomic SystemsStomachGastroenterologyMotilityAcetylcholineReceptors Neuropeptide YMice Inbred C57BL030104 developmental biologyEndocrinologymedicine.anatomical_structureMechanism of actionTachykininmedicine.symptomEnteric nervous systemGastrointestinal Motility030217 neurology & neurosurgeryAcetylcholinemedicine.drug
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The basic structural lesion of persistent neonatal hypoglycaemia with hyperinsulinism: deficiency of pancreatic D cells or hyperactivity of B cells?

1984

Pancreatic tissue obtained at subtotal pancreatectomy from 15 infants with persistent hypoglycaemia with hyperinsulinism, and autopsy specimens from 23 age-matched normoglycaemic controls, were studied with morphometric methods after immunocytochemical staining of the four main islet cell types (A, B, D and pancreatic polypeptide cells). In three cases, a focal lesion was detected by gross examination. Macroscopic or microscopic examination did not distinguish the 12 other cases from controls. As found previously, nesidioblastosis was not a specific feature of the pancreas in infantile hypoglycaemia, being observed in age-matched controls as well. In cases with hypoglycaemia the volume dens…

Malemedicine.medical_specialtyPancreatic Polypeptide-Secreting CellsCell typePathologyEndocrinology Diabetes and MetabolismNesidioblastosisCell CountLesionIslets of LangerhansInternal medicineHyperinsulinismInternal MedicinemedicineHumansB cellCell Nucleusbusiness.industryDegranulationInfant NewbornInfantmedicine.diseaseHypoglycemiaEndocrinologymedicine.anatomical_structureFemalemedicine.symptombusinessPancreasHyperinsulinismDiabetologia
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Neuropeptide Y receptors (version 2019.4) in the IUPHAR/BPS Guide to Pharmacology Database

2019

Neuropeptide Y (NPY) receptors (nomenclature as agreed by the NC-IUPHAR Subcommittee on Neuropeptide Y Receptors [156]) are activated by the endogenous peptides neuropeptide Y, neuropeptide Y-(3-36), peptide YY, PYY-(3-36) and pancreatic polypeptide (PP). The receptor originally identified as the Y3 receptor has been identified as the CXCR4 chemokine recepter (originally named LESTR, [137]). The y6 receptor is a functional gene product in mouse, absent in rat, but contains a frame-shift mutation in primates producing a truncated non-functional gene [83]. Many of the agonists exhibit differing degrees of selectivity dependent on the species examined. For example, the potency of PP is greater…

MutationChemokinebiologyChemistryNeuropeptidePharmacologymedicine.disease_causeNeuropeptide Y receptorPeptide YYmedicinebiology.proteinPancreatic polypeptideReceptorPeptide sequenceIUPHAR/BPS Guide to Pharmacology CITE
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VIPoma and PPoma

2015

Gastrinomas are rare tumors of the neuroendocrine system, occurring within the pancreas and duodenum. The annual incidence is estimated at 0.5 per million of the population [1]. Overproduction of gastrin by these tumors produces a sustained increase in gastric acid secretion, leading to clinical manifestations of complicated peptic ulcer disease known as the Zollinger-Ellison syndrome (ZES) [2]. Although most gastrinomas grow slowly, over 60 % are malignant and 25 % show rapid aggressive behavior [3-5]. Most commonly gastrinomas metastasize to regional lymph nodes and the liver, and it is this malignant potential which has become increasingly important since the introduction of effective me…

medicine.medical_specialtySettore MED/06 - Oncologia Medicabusiness.industryMedicine (all)Vasoactive intestinal peptidePlasma levelsmedicine.diseaseMalignancyGastroenterologyIslet cell tumourInternal medicineWatery diarrhoeaMedicinePancreatic polypeptideSecretionHuman medicinebusinessVIPoma
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